AllerGen researchers find possible key to fibrosis in mouse model of Crohn’s disease
New research by AllerGen investigators at The University of British Columbia (UBC) has found that a group of immune cells known as Group 3 innate lymphoid cells (ILC3) could be the key to the development of fibrosis in a mouse model of Crohn’s disease.
The discovery may be applicable to fibrosis in other types of tissue, and could eventually lead to the development of a treatment for fibrotic complications of Crohn’s disease.
Dr. Kelly McNagny, Associate Scientific Director of the AllerGen Network, is lead author on the study, published today (6 September 2016) in Science Immunology.